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In nerves uncovered to one hundred Hz when bathed in 10 mM glucose, the CAP failed suggesting that even within the presence of normoglycemic glucose further power substrate is required when the tissue power demand increases. Such knowledge are essential as they present that glycogen has an necessary position under physiological conditions. The truth that glycogen supported CAP conduction within the absence of exogenously utilized glucose in all probability has no value in ascribing a task to glycogen, since aglycaemia is an artificial experimental process, and hypoglycemia is an iatrogenic consequence of insulin therapy that did not exist prior to 1922 (Frier et al., 2014), thus can't be glycogen’s important position. However supplying lactate to axons when tissue vitality demand will increase implies a novel function for brain glycogen and suggests it acts as an vitality buffer rather than an power reserve, whose role is to not single-handedly assist conduction, but to act in live performance with ambient ranges of normoglycemic glucose.

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